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Ronald Kohanski

Deputy Director of the Division of Aging Biology at the National Institute on Aging.

Ronald Kohanski, PhD. is the Deputy Director of the Division of Aging Biology at the National Institute on Aging, NIH. Trained as a biochemist, he obtained a PhD in Biochemistry from the University of Chicago in 1981. After a postdoctoral fellowship with M. Daniel Lane at the Johns Hopkins University School of Medicine, he held a faculty position at the Mount Sinai School of Medicine for 17 years before returning as a faculty member at Johns Hopkins. His fields of research included enzymology and developmental biology of the insulin receptor. Dr. Kohanski joined the Division of Aging Biology, NIA in 2005 as a Program Officer, and became Division Deputy Director in 2007. Dr. Kohanski has promoted aging research in the specific areas of stem cell biology and cardiovascular biology. More broadly he promotes research efforts to expand studies beyond laboratory animals, to address the basic biology of aging explicitly in human populations and non-laboratory animals (domestic and wild populations).

Visit website: https://www.nia.nih.gov/about/staff/kohanski-ronald

See also: Institute National Institute on Aging (NIA) - Conducting and supporting research on aging and the health and well-being of older people

Details last updated 02-Nov-2019

Ronald Kohanski News

Super summary of 3rd Annual Longevity Therapeutics Summit

Super summary of 3rd Annual Longevity Therapeutics Summit

Lifespan.io (LEAF) - 16-Feb-2021

More and more companies making greater and greater progress - 2021 could be an exciting year

New research with senolytics shows promising results

New research with senolytics shows promising results

Longevity Technology - 25-Aug-2020

Senolytic research is in its nascent stage but no reason why one shouldn't be hopeful

Small Mayo Clinic trial successfully removes senescent cells from humans

Small Mayo Clinic trial successfully removes senescent cells from humans

Mayo Clinic - 18-Sep-2019

Occasional senolytic use may be enough to keep senescent cells at bay