Key points from article :
Decline of Cockayne Syndrome B protein in cells initiates cellular senescence.
Artificially reduced the amount of CSB protein in skin cells grown in laboratory cell culture.
Cells then stopped dividing but remained alive becoming senescent.
A compound called MnTBAP that mops up free radicals, delayed senescence for a limited time.
Blocked a part of the pro-senescent pathway downstream of CSB.
MnTBAP has the potential to become a medicine in the future, but needs more research at the moment.
Research by Institut Pasteur, published in Nature Communications.
