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Researchers discover how protein aggregation triggers aging

21-Apr-2017

Key points from article :

The insulin receptor acts like a brake on life expectancy.

Genetically altered animals where it no longer functions live much longer than normal.

Protein CHIP plays a crucial role in acting like a disposal helper.

When CHIP is missing, it leads to premature aging.

Unfortunatley if there is too much CHIP, undamaged proteins are also recycled and the organism is weakened.

Researchers by the universities of Cologne and Bonn published in the journal “Cell”.

Mentioned in this article:

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Cell

Scientific journal publishing research from many disciplines within the life sciences

Thorsten Hoppe

Principal Investigator in the Institute for Genetics at the University of Cologne.

University of Bonn

International research based university.

University of Cologne

University in Cologne, Germany.

Topics mentioned on this page:
Life Extension